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Fetzima Tapering Guide

levomilnacipran

SNRIFDA 2013
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Boxed Warning

Suicidality risk in children, adolescents, and young adults under 25 during initial treatment.

Overview

Levomilnacipran is the more pharmacologically active enantiomer of milnacipran. It is an SNRI approved for major depressive disorder with approximately 2:1 selectivity for norepinephrine over serotonin reuptake inhibition.

Common Doses

20mg, 40mg, 80mg, 120mg

Formulations

Extended-release capsules: 20mg, 40mg, 80mg, 120mg

Pregnancy

Category C (risk cannot be ruled out)

Mechanism of Action

Potent dual reuptake inhibitor of serotonin and norepinephrine with preferential norepinephrine activity (NET > SERT, approximately 2:1 ratio). The active S-enantiomer of milnacipran.

Taper Notes

Active S-enantiomer of milnacipran with preferential norepinephrine reuptake inhibition (NET:SERT ~2:1). Once-daily ER formulation. Discontinuation syndrome similar to other SNRIs; taper gradually over weeks using available capsule strengths.

Maudsley Deprescribing Guidance

Step down by one dose level (e.g., 120 → 80 → 40 → 20 mg) every 1–2 weeks. Do not crush or open ER capsules — matrix integrity is required for controlled release.

Common Withdrawal Symptoms

dizzinessnauseaheadacheirritabilityinsomniafatiguehyperhidrosis

Interactions & Safety

Drug Interactions

  • MAOIs — contraindicated (risk of serotonin syndrome)
  • Strong CYP3A4 inhibitors (ketoconazole) — max dose 80mg/day
  • Serotonergic drugs increase serotonin syndrome risk

Food Interactions

  • No significant food effect on absorption
  • May be taken with or without food

Contraindications

  • MAOIs within 14 days
  • Uncontrolled narrow-angle glaucoma
  • Known hypersensitivity to levomilnacipran or milnacipran

Toxicity

Serotonin syndrome risk. Blood pressure and heart rate increases. Urinary hesitation.

Pharmacokinetics

ADME Profile

Absorption

Well absorbed, bioavailability ~92%. Tmax ~6–8 hours (extended-release). Food does not affect AUC.

Distribution

387–473 L

Metabolism

Hepatic via CYP3A4 (major) with minor contribution from CYP2C8, CYP2C19, CYP2D6, and CYP2J2. Desethyl metabolite (inactive).

Elimination

Renal (58% unchanged).

Protein Binding

22%

Clearance

~21 L/hr

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