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Viibryd Tapering Guide

vilazodone

SSRIFDA 2011
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Boxed Warning

Suicidality risk in children, adolescents, and young adults under 25 during initial treatment.

Overview

Vilazodone is a selective serotonin reuptake inhibitor (SSRI) and partial agonist at the 5-HT1A receptor. It is used for the treatment of major depressive disorder.

Common Doses

10mg, 20mg, 40mg

Formulations

Tablets: 10mg, 20mg, 40mg

Pregnancy

Category C (risk cannot be ruled out)

Mechanism of Action

Combined SSRI activity with 5-HT1A receptor partial agonism, providing dual serotonergic modulation. Blocks SERT while partially activating 5-HT1A autoreceptors.

Taper Notes

Dual mechanism (SSRI + 5-HT1A partial agonist) and food-dependent absorption (~50% reduction without food) complicate tapering. No liquid formulation available; rely on tablet splitting. GI rebound is the predominant withdrawal pattern.

Maudsley Deprescribing Guidance

Reduce gradually over months. Initial 10 mg steps using available tablet strengths, transitioning to proportional ~10% reductions at lower doses. Compounded suspension may be needed for terminal doses given lack of commercial liquid.

Common Withdrawal Symptoms

dizzinessnauseadiarrheainsomniaheadacheirritability

Interactions & Safety

Drug Interactions

  • MAOIs — contraindicated (risk of serotonin syndrome)
  • Strong CYP3A4 inhibitors (ketoconazole) — reduce vilazodone dose to 20mg
  • Serotonergic drugs (triptans, tramadol, St. John's Wort) increase serotonin syndrome risk

Food Interactions

  • MUST be taken with food — bioavailability drops ~50% without food
  • No specific food contraindications beyond the requirement to take with food

Contraindications

  • MAOIs within 14 days
  • Known hypersensitivity to vilazodone

Toxicity

Serotonin syndrome risk when combined with serotonergic agents. GI side effects are the most common dose-limiting toxicity.

Pharmacokinetics

ADME Profile

Absorption

Oral bioavailability 72% with food (significantly reduced without food). Tmax ~4–5 hours. Must be taken with food.

Distribution

Not well characterized; extensively distributed.

Metabolism

Hepatic via CYP3A4 (major), CYP2C19, and CYP2D6. Active metabolites are not clinically significant.

Elimination

Primarily fecal (2% unchanged in urine).

Protein Binding

96–99%

Clearance

Apparent clearance ~40 L/hr

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